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Programmed cell death

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  1. Introduction
  2. The potential of Horvitz's research
  3. Programmed cell death in C. Elegans
  4. The process of programmed cell death
  5. The two parallel pathways signal transduction
  6. The process of engulfment
  7. The final step in programmed cell death
  8. Conclusion

H. Robert Horvitz won the 2002 Nobel Prize in Physiology or Medicine for his work on programmed cell death (apoptosis) in the Caenorhabditis elegan roundworm. Horvitz discovered that cells don't die because they have been damaged by life or outside environment, but rather it is caused by an active biological process of programmed cell death or cell suicide. Previously, it was debated if programmed cell death was an active biological process, but Horvitz research explains that programmed cell death happens as frequently as cell division, morphogenesis and cell differentiation that every cell goes through.

[...] Programmed cell death H. Robert Horvitz won the 2002 Nobel Prize in Physiology or Medicine for his work on programmed cell death (apoptosis) in the Caenorhabditis elegan roundworm. Horvitz discovered that cells don't die because they have been damaged by life or outside environment, but rather it is caused by an active biological process of programmed cell death or cell suicide. Previously, it was debated if programmed cell death was an active biological process, but Horvitz research explains that programmed cell death happens as frequently as cell division, morphogenesis and cell differentiation that every cell goes through. [...]


[...] The process of engulfment is an important part of programmed cell death, and more knowledge of this stage of programmed cell death can lead to disease intervention. Using different cells to either block or cause the process of program cell death can have a positive or negative effect on degenerative diseases. Further research discovered that CED-3 has two enhancer partial killer genes defined by the mutations of N3376 and N3380. N3376 encodes a novel Zn finger protein called MCD-1, and n3380 is an allele of DPL-1. [...]

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