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LED syndrome antiphospholipide and CNS

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  1. Summary
  2. Introduction
  3. Neuropsychiatric manifestations and LED
  4. Neurological manifestations of primary LED
  5. Psychiatric manifestations of lupus primary brain
  6. Diagnostic approach of neuropsychiatric manifestations of systemic lupus erythematosus
  7. Brain imaging
  8. Cytokines, polymorphisms and LED
  9. HLA and cytokines
  10. Conclusion
  11. Bibliography

The neurolupus represents the second leading cause of death in the LED. Although its incidence is high, it remains difficult to diagnose because of the diversity of events and the possible confusion with other brain diseases. In the absence of pathognomonic lesions, the diagnostic approach based on research criteria for an LED, the distinction between functional or organic etiologies and the exclusion of symptoms unrelated to the LED. The search for specific serological parameters, an analysis of the LCR, an EEG, a brain imaging psychomotor tests or brain biopsy, if necessary, will be carried out. If the CNS lesions are associated with a syndrome antiphospholipide, treatment is based on anticoagulation otherwise immunosuppression should be considered

[...] Neuropsychiatric manifestations and LED It should be a distinction between neuropsychiatric events by forming part of the basic disease and may possibly occur as initial symptoms of SLE, other complications secondary disease once established. Neuropsychiatric manifestations of LEDs can be classified into neurological and psychiatric manifestations primary and secondary events. Their presentation may be diffuse, in which case they are often transient, reversible treatment and association with a specific pathological finding; unlike focal manifestations, often presenting acute refractory to treatment and for which histological lesions are found at autopsy. [...]

[...] Anxiety in the setting of the diagnosis LED or during exacerbations of the disease is manifested by palpitations, diarrhea, sweating, hyperventilation, difficulty speaking, memory impairment, and headache. The risk of progression to obsessive compulsive behavior, phobias or hypochondriac disorders should be considered. Diagnostic approach of neuropsychiatric manifestations of systemic lupus erythematosus There is no specific test to establish to establish the diagnosis of lupus neuropsychia tion. The diagnosis is, therefore, to establish a first step in the diagnosis of systemic lupus erythematosus and then seeks to distinguish attacks organic functional brain damage, and to exclude symptoms not related to an LED. [...]

[...] Other factors including Ac antibodies of metabolic disorders, hypertension, infections, neoplasms, stroke, and withdrawal of medication, a vasculopathies or drug toxicity (antimalarials) have been described. Complex partial seizures are often an initial manifestation of SLE, they are highly correlated with the presence of a psychosis of paranoid type. Epilepsy in lupus patients is a marker of poor pronostic. The cephalees are frequent complaints. Many etiologies are possible: Because of the tendency of depression and anxiety caused by illness, Cepha voltage-tion have an increased prevalence in lupus. [...]

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