In order for strenuous exercise to continue, high-energy phosphates must be resynthesised at a rapid rate (McArdle et al). Glycogen stored in the muscle is the main source of energy during intense exercise. This results in the phosphorylation of ADP through anaerobic glycolysis and leads to the formation of lactate. According to McArdle et al, rapid, large accumulations of blood lactate occur during maximal exercise. Decreasing the intensity of the exercise will decrease the rate of accumulation and the final level of blood lactate.
[...] The subject's blood lactate was measured in the last 30 sec of each stage. Heart rate, RPE (perceived exertion), RER (resting energy expenditure), VO2 and VCO2 were also measured at each stage. The test continued until the subject had reached their max; this was at 175 watts or 1050 KGM. The test was terminated and the blood lactate was measured immediately and the subject cooled down by cycling at her own pace for about 2 mins. Results The subject was 22-year-old Sarah. [...]
[...] The purpose of this lab was to examine variations in blood lactate during rest and various exercise intensity. Methods The cycle ergometer was used in this lab. The first thing was to calibrate the metabolic cart. This was done by entering volumes of air into the computer, using a 3L syringe. The calibration was done continuously until the sensor read 3L. Next, the percentages of O2 and CO2 were calibrated by running the known concentrations of mixed gases into the gas analyzer. [...]
[...] The gas exchange threshold using this method was This value is almost similar to the value from the V-slope method, which was Conclusions The subject reached her LT at about of her max. This shows that the subject was classified as healthy because according to McArdle et al in healthy people this should occur anywhere between 50- of ones max. If the subject were a trained athlete, the LT would have shifted to the right and would have occurred at about 80-90% of her max. [...]
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